pars compacta

The pars compacta is heavily involved in learned responses to stimuli.

The name reticulata is simply an opposition to the dense pars compacta located above it.

The pars compacta is a portion of the substantia nigra.

The pars compacta also plays a role in temporal processing and is activated during time reproduction.

The pars compacta is also important in spatial learning, the observations about one's environment and location in space.

Lesions in the pars compacta leads to temporal deficits.

The cause of death of dopaminergic neurons in the pars compacta is unknown.

However, some contributions to the unique susceptibility of dopaminergic neurons in the pars compacta have been identified.

In humans, the nerve cell bodies of the pars compacta are coloured black by the pigment neuromelanin.

Parkinson's disease is the slow and steady loss of dopaminergic neurons in substantia nigra pars compacta.

The motor role of the pars compacta may involve fine motor control, as has been confirmed in animal models with lesions in that region.

The dopamine neurons are found mainly in a part of this structure called the pars compacta (cell group A8) and nearby (group A9).

Another well known afferent is the nigrostriatal connection arising from the neurons of the substantia nigra pars compacta.

Gene expression patterns in the adult mouse show high expression in the substantia nigra pars compacta.

The two pallidal nuclei receives dopaminergic axons from the pars compacta of the substantia nigra.

The pars compacta serves mainly as an input to the basal ganglia circuit, supplying the striatum with dopamine.

It is for the most part identical with the pars compacta of the substantia nigra as defined on the basis of Nissl stains.

MPP+ kills primarily dopamine-producing neurons in a part of the brain called the pars compacta of the substantia nigra.

Secondly, dopaminergic neurons in the pars compacta contain less calbindin than other dopaminergic neurons.

They make connections with the dopamine neurons of the pars compacta whose long dendrites plunge deeply in the pars reticulata.

The long ventral dendrites of the pars compacta indeed plunge deep in the pars reticulata where they receive synapses from the bundle.

The inhibition of dopamine reuptake by cocaine also inhibits the firing of spontaneous action potentials by the pars compacta.

Pathologically, it is characterized by the loss of dopaminergic cells of the substantia nigra, pars compacta (SNc).

The antagonistic functions of the direct and indirect pathways are modulated by the substantia nigra pars compacta (SNc), which produces dopamine.

Parkinson's disease is usually associated with a degraded substantia nigra pars compacta, but recent evidence suggests that PD affects a number of sites in the brain.