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Lytic infection is sometimes known as "productive" infection.
Herpesviridae can cause latent or lytic infections.
The virus also causes lytic infection in the oropharynx, but is kept in check by a normal, functioning immune system.
In vivo , it would be difficult to pinpoint one specific factor to cause the switch from latency to lytic infection.
Uncontrolled lytic infection is manifested as oral hairy leukoplakia in immunocompromised hosts.
Once the virus's initial lytic infection is brought under control, EBV latently persists in the individual's B cells for the rest of the individual's life.
Also, the repressor produced by the prophage that prevents prophage genes from being repressed confers an immunity for the host bacteria from lytic infection by related viruses.
Latency is distinguished from lytic infection; in lytic infection many Herpes virus particles are produced and then burst or lyse the host cell.
HHV Infected Cell Polypeptide 0 (ICP0) gene is expressed very early during lytic infection, and for this reason is called an immediate-early Herpes gene.
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