locus coeruleus , także: locus cinereus

The locus coeruleus may figure in clinical depression, panic disorder, and anxiety.

The locus coeruleus is responsible for mediating many of the sympathetic effects during stress.

These regions are called locus coeruleus and the lateral tegmental.

"A brain structure as significant as the locus coeruleus is bound to have an effect on personality."

Degeneration is also present in brainstem nuclei like the locus coeruleus.

Norepinephrine, which is involved in arousal, comes exclusively from a nearby small area called the locus coeruleus.

Opioids inhibit the firing of neurons in the locus coeruleus.

While the heightened stress response is likely to involve the thalamus, hypothalamus and locus coeruleus.

The locus coeruleus is the principal site for brain synthesis of norepinephrine (noradrenaline).

The locus coeruleus receives inputs from a number of other brain regions, primarily:

Recent autopsy studies have shown that in people between the ages of 40 and 60, the locus coeruleus undergoes a sharp deterioration.

The locus coeruleus is tiny; it has just 20,000 or so cells whereas most brain structures have millions of cells.

As an important homeostasis control center of the body, the locus coeruleus receives afferents from the hypothalamus.

The noradrenaline system consists of just 1500 neurons on each side of the brain, primarily in the locus coeruleus.

Norepinephrine is a neurotransmitter released from locus coeruleus when stimulated by the hypothalamus during a stress response.

Norepinephrine can also suppress neuroinflammation when released diffusely in the brain from the locus coeruleus.

The noradrenergic neurons originate both in the locus coeruleus and the lateral tegmental field.

Eventually the neurons in the locus coeruleus enter a refractory period, due to the auto-inhibitory effect of norepinephrine.

Stimulation of the locus coeruleus increases noradrenaline release mediating physiological and behavioural arousal.

The pathways begin in the locus coeruleus (noradrenaline) and the nucleus of raphe (serotonin).

Transmission of noradrenaline from the caudal raphe nuclei and the locus coeruleus is increased in phobia.

When opioid consumption is stopped, the increased activity of the locus coeruleus contributes to the symptoms of opiate withdrawal.

These Locus Coeruleus cell changes include hyperexcitability and decreased functioning of its noradrenergic innervation.

Physiological symptoms of the panic response are medicated by the autonomic nervous system through connections with the locus coeruleus and hypothalamus.

In panic disorder there is increased norepinephrine transmission from both the locus coeruleus and the caudal raphe nuclei.